References |
"Moderate loss of function of cyclic-AMP-modulated KCNQ2/KCNQ3 K+channels causes epilepsy.";,Schroeder B.C., Kubisch C., Stein V., Jentsch T.J.;,Nature 396:687-690(1998)., PubMeb:9872318 DOI:10.1038/25367
"A pore mutation in a novel KQT-like potassium channel gene in anidiopathic epilepsy family.";,Charlier C., Singh N.A., Ryan S.G., Lewis T.B., Reus B.E., Leach R.J.,Leppert M.;,Nat. Genet. 18:53-55(1998)., PubMeb:9425900 DOI:10.1038/ng0198-53
"Functional expression of two KvLQT1-related potassium channelsresponsible for an inherited idiopathic epilepsy.";,Yang W.-P., Levesque P.C., Little W.A., Conder M.L., Ramakrishnan P.,Neubauer M.G., Blanar M.A.;,J. Biol. Chem. 273:19419-19423(1998)., PubMeb:9677360 DOI:10.1074/jbc.273.31.19419
"Two types of K(+) channel subunit, Erg1 and KCNQ2/3, contribute tothe M-like current in a mammalian neuronal cell.";,Selyanko A.A., Hadley J.K., Wood I.C., Abogadie F.C., Delmas P.,Buckley N.J., London B., Brown D.A.;,J. Neurosci. 19:7742-7756(1999)., PubMeb:10479678
"M-type KCNQ2-KCNQ3 potassium channels are modulated by the KCNE2subunit.";,Tinel N., Diochot S., Lauritzen I., Barhanin J., Lazdunski M.,Borsotto M.;,FEBS Lett. 480:137-141(2000)., PubMeb:11034315 DOI:10.1016/S0014-5793(00)01918-9
"Surface expression and single channel properties of KCNQ2/KCNQ3, M-type K+ channels involved in epilepsy.";,Schwake M., Pusch M., Kharkovets T., Jentsch T.J.;,J. Biol. Chem. 275:13343-13348(2000)., PubMeb:10788442 DOI:10.1074/jbc.275.18.13343
"Reconstitution of muscarinic modulation of the KCNQ2/KCNQ3 K(+)channels that underlie the neuronal M current.";,Shapiro M.S., Roche J.P., Kaftan E.J., Cruzblanca H., Mackie K.,Hille B.;,J. Neurosci. 20:1710-1721(2000)., PubMeb:10684873
"Inhibition of KCNQ1-4 potassium channels expressed in mammalian cellsvia M1 muscarinic acetylcholine receptors.";,Selyanko A.A., Hadley J.K., Wood I.C., Abogadie F.C., Jentsch T.J.,Brown D.A.;,J. Physiol. (Lond.) 522:349-355(2000)., PubMeb:10713961DOI:10.1111/j.1469-7793.2000.t01-2-00349.x
"Modulation of KCNQ2/3 potassium channels by the novel anticonvulsantretigabine.";,Main M.J., Cryan J.E., Dupere J.R., Cox B., Clare J.J., Burbidge S.A.;,Mol. Pharmacol. 58:253-262(2000)., PubMeb:10908292
"Retigabine, a novel anti-convulsant, enhances activation of KCNQ2/Q3potassium channels.";,Wickenden A.D., Yu W., Zou A., Jegla T., Wagoner P.K.;,Mol. Pharmacol. 58:591-600(2000)., PubMeb:10953053
"The novel anticonvulsant retigabine activates M-currents in Chinesehamster ovary-cells tranfected with human KCNQ2/3 subunits.";,Rundfeldt C., Netzer R.;,Neurosci. Lett. 282:73-76(2000)., PubMeb:10713399 DOI:10.1016/S0304-3940(00)00866-1
"Characterization of KCNQ5/Q3 potassium channels expressed inmammalian cells.";,Wickenden A.D., Zou A., Wagoner P.K., Jegla T.;,Br. J. Pharmacol. 132:381-384(2001)., PubMeb:11159685 DOI:10.1038/sj.bjp.0703861
"Improved titanium dioxide enrichment of phosphopeptides from HeLacells and high confident phosphopeptide identification by cross-validation of MS/MS and MS/MS/MS spectra.";,Yu L.-R., Zhu Z., Chan K.C., Issaq H.J., Dimitrov D.S., Veenstra T.D.;,J. Proteome Res. 6:4150-4162(2007).,PubMeb:17924679 DOI:10.1021/pr070152u
"A novel mutation of KCNQ3 (c.925T-->C) in a Japanese family withbenign familial neonatal convulsions.";,Hirose S., Zenri F., Akiyoshi H., Fukuma G., Iwata H., Inoue T.,Yonetani M., Tsutsumi M., Muranaka H., Kurokawa T., Hanai T., Wada K.,Kaneko S., Mitsudome A.;,Ann. Neurol. 47:822-826(2000)., PubMeb:10852552DOI:10.1002/1531-8249(200006)47:6<822::AID-ANA19>3.3.CO2-O;
"KCNQ2 and KCNQ3 potassium channel genes in benign familial neonatalconvulsions: expansion of the functional and mutation spectrum.";,Singh N.A., Westenskow P., Charlier C., Pappas C., Leslie J.,Dillon J., Anderson V.E., Sanguinetti M.C., Leppert M.F.;,Brain 126:2726-2737(2003).,PubMeb:14534157 DOI:10.1093/brain/awg286 |
